Corrigendum to “Long-Term Nitric Oxide Exposure Enhances Lung Cancer Cell Migration”

نویسندگان

  • Arpasinee Sanuphan
  • Preedakorn Chunhacha
  • Varisa Pongrakhananon
  • Pithi Chanvorachote
چکیده

In the paper titled " Long-Term Nitric Oxide Exposure Enhances Lung Cancer Cell Migration, " the Acknowledgments is corrected as follows.

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منابع مشابه

Long-Term Nitric Oxide Exposure Enhances Lung Cancer Cell Migration

Nitric oxide (NO) found in the vicinity of lung cancer cells may play a role in the regulation of cancer cell behaviors. To explore the possible effects of NO on cell motility, human lung cancer cells were exposed to nontoxic concentrations of NO for 0-14 days, and the migratory characteristics of the cells were determined. The present study found that long-term treatment with NO significantly ...

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Two Lung Cancer Development-Related Genes, Forkhead Box M1 (FOXM1) and Apolipoprotein E (APOE), are overexpressed in Bronchial of Patients after Long-Term Exposure to Sulfur Mustard

Sulfur mustard (SM) is a strong alkylating and mutagenic compound that targets humanairway system. We considered the expression of Forkhead box M1 (FOXM1) and apolipoproteinE (APOE) genes, which are responsible for cell proliferation, differentiation, tumorigenesis,and increased risk of lung cancer, in the lung bronchial tissue of patients exposed to SM.After performing pulmonary functional tes...

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Two Lung Cancer Development-Related Genes, Forkhead Box M1 (FOXM1) and Apolipoprotein E (APOE), are overexpressed in Bronchial of Patients after Long-Term Exposure to Sulfur Mustard

Sulfur mustard (SM) is a strong alkylating and mutagenic compound that targets humanairway system. We considered the expression of Forkhead box M1 (FOXM1) and apolipoproteinE (APOE) genes, which are responsible for cell proliferation, differentiation, tumorigenesis,and increased risk of lung cancer, in the lung bronchial tissue of patients exposed to SM.After performing pulmonary functional tes...

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Prolonged Nitric Oxide Exposure Enhances Anoikis Resistance and Migration through Epithelial-Mesenchymal Transition and Caveolin-1 Upregulation

Nitric oxide (NO) in tumor microenvironment may have a significant impact on metastatic behaviors of cancer. Noncytotoxic doses of NO enhanced anoikis resistance and migration in lung cancer H23 cells via an increase in lamellipodia, epithelial-mesenchymal transition (EMT) markers including vimentin and snail, and caveolin-1 (Cav-1). However, the induction of EMT was found in Cav-1-knock down c...

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Acquired resistance to chemotherapy in lung cancer cells mediated by prolonged nitric oxide exposure.

BACKGROUND The effect of extended exposure of cancer cells to nitric oxide (NO), an endogenous mediator frequently found increased in tumors, is largely unknown. In the present study, the effect of long-term NO exposure on chemotherapeutic resistance was investigated in lung cancer cells. MATERIALS AND METHODS The effect of long-term exposure of human lung cancer cells to NO on susceptibility...

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عنوان ژورنال:

دوره 2015  شماره 

صفحات  -

تاریخ انتشار 2015